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Enzymatic degradation

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Oxygenation of the perfusate promotes intracellular ATP synthesis and delays ischemic injury, which is known as the HMPox protocol (1, 140, 141)

Intriguingly, we observed that glr1 cells had an extended lag phase, while the additional deletion of PRX1 ( glr1 prx1 cells) rescued this growth delay (Fig 7B)