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The mechanism likely involves correction of oxidative stress and modulation of glutamate signaling via the cystine-glutamate antiporter (system Xc-) [73][74]
This is the clinical standard that protects you from the unverified compounds prevalent in the unregulated peptide market
& Lavie, A
Therefore, sufficient GSH supply and active glutathione peroxidase are main defence resources of neurons under pro-oxidative conditions 23,24,25
Oxidative damage has been widely implicated in this disease, though the mechanisms involved remain elusive
