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Derks NAV, Krugers HJ, Hoogenraad CC, Jels M, Sarabdjitsingh RA
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Combined with morphological observations, these transcriptional data allow us to propose a model for parasitic responses to treatment: Following DHA exposure, parasites at early stages upregulate protein synthesis to repair DHA-induced damagea compensatory mechanism that ultimately fails, as DHA causes irreversible, lethal injury
J Endocrinol 2014;221:2937
BPC-157s healing effects are thought to be driven by several interconnected biological mechanisms involving nitric oxide (NO) modulation, growth factor signaling, angiogenesis, and cellular repair pathways, although most evidence remains preclinical
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