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Given the robustness of this mitochondrial response, the multifaceted nature of PGC1 regulation in cells and a modest rescue at best by mTORC1 inhibition, we suspect that there is a degree of functional redundancy built into this metabolic plasticity system that can contribute to such an adaptive response, and complete inhibition of this system will prove difficult
Afamelanotide and melanotan-1 are the same molecule (Nle4-D-Phe7-alpha-MSH) with different names, regulatory statuses, and delivery systems
Microsurgery 42 (5), 433440
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