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Multiple roles of the ERCC1-XPF endonuclease in DNA repair and resistance to anticancer drugs

The ROR with 95% CI was calculated as: In these equations, a represents the number of reports with both target drug exposure and target AEs

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[27] The connection between GSTP inhibition of the pro-apoptotic JNK pathway and the isozyme's overexpression in drug-resistant tumor cells may itself account for the tumor cells' ability to escape apoptosis mediated by drugs that are not substrates of GSTP