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13 (Suppl 1), S6568 (1992)

Reviewed by Esther A Torres MD, AGAF Professor of medicine, University of Puerto Rico School of Medicine, San Juan Esther A Torres MD, AGAF Professor of medicine, University of Puerto Rico School of Medicine, San Juan

Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants

Mitochondrial dysfunction causes ROS accumulation and activates inflammatory pathways, releasing pro-inflammatory cytokines and inflammasomes

S1 are freely accessible at and only specific data of Gst gene expression are summarized in Table S3

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