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Roles of the glutathione- and thioredoxin-dependent reduction system in the Escherichia coli and Saccharomyces cerevisiae responses to oxidative stress

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Our findings of decreased NRF2 levels seem to contradict previous studies suggesting an increased NRF2 response in Down syndrome

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Finally, we used to model to show how the observed metabolic profile of Down syndrome could arise from trisomy 21 and moderate oxidative stress and how the observed metabolic profile of autism patients could arise from high oxidative stress and increased adenosine concentration