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These data suggest that while markers of fibrosis and autophagy are increased following liver-specific Gclc deletion, these are potentially due to the activation of NRF2
doi: 10.1186/1743-7075-8-53 247
An immunodominant and potentially pathogenic epitope associated with these blistering diseases has recently been mapped to the extracellular domain of a human epidermal antigen, BP180
Useful markers for senescence biology in a research context include hs-CRP, IL-6, and IGF-1, alongside a comprehensive metabolic panel
M.HwangS
Metabolic Dysfunction in Alzheimer's Disease and Related Neurodegenerative Disorders
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