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Recruitment of cBAF and PBAF complexes is required for transcription of SLC7A11 SLC7A11 To determine the mechanism underlying the attenuation of SLC7A11 mRNA expression in SWI/SNF-deficient cancer cells, we investigated the involvement of SMARCA4 (a component of cBAF, PBAF, and ncBAF) and SMARCB1 (a component of cBAF and PBAF) in transcriptional upregulation of SLC7A11

Neutron spectroscopy and MD simulations are also regularly used in dynamic studies of lipid-protein interactions due to the compatibility of the length and time scales that they can access