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This origin suggests GHK-Cu peptide may function as an extracellular damage signal, potentially interacting with cell-surface receptors, ion channels, and intracellular enzymes to coordinate repair-associated responses

Dihexa binds to HGF with high affinity and potentiates HGF's ability to activate c-Met at concentrations far below normal thresholds

A parallel phenotype was observed in paraquat-treated fly model where Sp administration resulted in lower accumulation of oxygen radicals in mitochondria of brain neurons (Fig