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In healthy tissue, BPC-157 appears to maintain nitric oxide homeostasis by modestly increasing eNOS activity while suppressing iNOS
Activating Transcription Factor 3 (ATF3, and inducible nitric oxide synthase (iNOS) has been shown to impact on extracellular matrix biosynthesis through increased collagen synthesis (Wang et al., 2021b)
doi: 10.1136/gutjnl-2018-316723 24 SorbaraMTPamerEG
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TB-500 and BPC-157 both promote angiogenesis, which theoretically could support tumor growth
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