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Liraglutide also induction HO-1 expression by activating AMPK and promoting HIF-1 protein export from cytoplasm to nucleus, which significantly increases the expression of anti-apoptotic protein Bcl-2 and decreases the expression of pro-apoptotic Bax and Caspase-3 in HUVECs cells [134]
There is thus a need for robust tools that can quantify this critical redox modulator with subcellular specificity
2011;3(78):78ra32
Certain metabolic intermediates derived from GSH catabolism can enter the tricarboxylic acid (TCA) cycle, thereby supporting mitochondrial ATP production (7)
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