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doi: 10.1136/adc.9.52.251
Endothel Dysfunct - Old Concepts New Chall [Internet]
mTORC1, which is well known for its function at the center of cancer metabolic reprogramming, promotes mitochondrial glutaminolysis via the migration of SIRT4-mediated inhibition of GLUD1 189
Baseline eGFR and UACR values in these studies suggest that participants were not predominantly at high-risk for CKD progression [80,81]
Beyond glycemic control: GLP-1 receptor agonists and their impact on calcium homeostasis in real-world patients
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