fsp1 is a glutathione-independent ferroptosis suppressor FSP1: key regulator of ferroptosis: Trends in Molecular Medicine Identification and characterization of FSP1

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The enhanced hepatic mitochondrial activity in the context of TKT deficiency has been shown to be the result of increased phospholipid synthesis, particularly increased synthesis of phosphatidylcholine which represents the major phospholipid in the mitochondrial membrane