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Presented for research literature review only Kou X et al
In laboratory animals, the forced overexpression of the TKT gene promotes the development of MAFLD as well as the advancement of MAFLD to the more severe state referred to as metabolic dysfunction-associated steatohepatitis (MASH)
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doi: 10.1007/s00204-019-02621-4 Summary Keywords herbal therapy, acetaminophen, liver injury, P450 enzyme, oxidative stress, gut microbiota Citation Chang L, Xu D, Zhu J, Ge G, Kong X and Zhou Y (2020) Herbal Therapy for the Treatment of Acetaminophen-Associated Liver Injury: Recent Advances and Future Perspectives
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Protein kinase C-mediated heat shock protein beta-1 (HSPB1) phosphorylation can stabilize the actin cytoskeleton, thereby inhibiting TfR1-mediated iron uptake and reducing lipid ROS production to limit ferroptotic cell death [54]
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