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The role of endogenous amylin as a satiating agent is supported by the observation that injection of the amylin receptor antagonist AC187 intravenously or directly into the area postrema acutely increases food intake in rats (47, 48)
Our findings build on previous research showing high discontinuation rates, particularly among individuals using GLP-1 RAs for weight management ( 10 ), and provide new insights into how and why these medications are stopped in real-world clinical settings
Targeting of the PI3K/AKT/GSK3 pathway in parkinson's disease: a therapeutic blueprint
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