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Association of traumatic brain injury with late-life neurodegenerative conditions and neuropathologic findings
NAFLD patients can effectively use oral supplementation at 300 mg daily without IV administration
The activation of GLP-1R can reduce the size of cerebral infarction by enhancing cell survival signaling pathways, reducing ischemia-reperfusion injury, promoting brain repair, and inhibiting inflammatory response and oxidative stress (8588)
The error has been fixed in the paper
Postprandial gallbladder emptying in patients with type 2 diabetes: potential implications for bile-induced secretion of glucagon-like peptide 1
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