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And it is asking us to retire the assumption that a drug that works for most people works for everyone

Greater insulin release with exendin-phe1 was unlikely to result from cross-reactivity with other incretin receptors, as it was blocked with the GLP-1R orthosteric antagonist exendin(9-39) (Supplementary Fig

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This chemical modification, developed by Theratechnologies, protects the peptide from degradation by dipeptidyl peptidase IV (DPP-IV), the main enzyme responsible for the short half-life of native GHRH

doi: 10.1186/s12890-021-01422-9 292 WestA