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Cell Cycle 15:559572
Targeting ferroptosis as a vulnerability in pulmonary diseases
Expert Featured In This Story Stuart Phillips Professor Stuart Phillips is a professor of Kinesiology and the Tier 1 Canada Research Chair in Skeletal Muscle Health
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If the ratio falls too low, the highly oxidized environment triggers cell cycle arrest or programmed cell death (apoptosis), leading to widespread tissue damage and profound fatigue
While the mechanism of action for BPC-157 is still unclear, it is believed to entail the upregulation of growth factors, the modulation of nitric oxide (NO) synthesis, and the control of functions of collagen fragments that are associated with bone morphogenic proteins [1]
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