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This underscores the possibility of modulating ferroptosis as an approach to counteract drug resistance and enhance therapeutic efficacy

The side effect profiles of cagrilintide and retatrutide are broadly consistent with other incretin-based therapies, reflecting their shared mechanisms of action

This is different from broad anti-inflammatory compounds that suppress everything

However, some reports have suggested that KD may not improve lipid metabolism and could potentially exacerbate dyslipidemia, leading to a pro-inflammatory state [77]

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