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One major concern is that chronic use of GLP-1 RAs may create a supraphysiological state of GLP-1, leading to gastrointestinal upset, pancreatitis, or excessive stimulation of pancreatic activity

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pylori infection is known to stimulate ROS production [25,3335], which could in principle deplete intracellular pools of reduced GSH by generating the oxidized disulfide dimer GSSG

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By periodically suppressing mTOR, rapamycin enhances autophagy (cellular cleanup), improves immune function, and targets the fundamental mechanisms of aging at the cellular level