Variables such as advanced age, poor initial neurological state, large hematoma volume, and intraventricular extension are independent predictors of poor outcomes following spontaneous ICH 5,6,7,8
For now, the best path forward is to stick with therapies backed by solid, long-term data, using GLP-1 agonists as a potential supplement for managing metabolic issues
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This compares favorably to semaglutide 2.4 mg, which reduced hepatic fat by approximately 40% in the NASH-CRN substudy of STEP-1
Thirdly, both drugs modulate metabolic parameters, such as blood pressure, and uric acid, and hence exert multiple metabolic benefits to various organs in the body, including the heart, brain, liver, pancreas, and kidney
15,52 Moreover, it has been suggested that there is a correlation between cortical atrophy, white matter hyperintensities, and hypometabolism with neurological complaints of patients