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1L), validating Liuyang et al.s chemical reprogramming protocol while extending its application to multi-system ASD pathology [27]
These data suggest that while markers of fibrosis and autophagy are increased following liver-specific Gclc deletion, these are potentially due to the activation of NRF2
doi: 10.1007/s12011-023-03782-1 81
322 Another study by Jeong et al
Simonsen laboratories, Gilroy, CA)
The lactate transported from astrocytes into neurons plays a crucial role in activating signaling cascades via MCT2 and HCAR1 (GPR81) receptors, involving plasticity-associated genes [183, 184]
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