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Not only do we lose the ability to repair, but we also lose the ability to correctly rebuild damaged tissues

More importantly, it is not an approved medicine anywhere in the world

Safety data is still limited, since most studies are small and short-term

The two can be combined, as they work through different mechanisms

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Even if it is still unclear whether OS is a primary or a secondary cause of neurodegeneration in ALS, overwhelming evidence indicates the pathological role of OS, and alterations in KEAP1 and NRF2 expression and in KEAP1-NRF2 activity may explain the progressive MN degeneration and death associated with ALS [170]