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J Clin Invest 1993;92:30814

However, in heterodimeric GST complexes, such as those formed by cytoplasmic mu and , the gap between the two subunits is the location of another high-affinity non-substrate heterologous biological binding site This may explain the ability of the enzyme to form heterodimers

62,109 These examples demonstrate that LPO and ferroptosis may be biologically irrelevant for APAP hepatotoxicity under normal conditions because endogenous defense mechanisms (such as the lipid-soluble antioxidant vitamin E and the iron chelator ferritin) effectively prevent excessive LPO

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Tirzepatide: dual-pathway incretin research compound with broad vial-size coverage