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doi: 10.1159/000497123, 14 Brozek-MuchaZ.ZdebK
Many investigators benefit from examining a panel of related compounds in parallel, as this approach can reveal differential effects and structure-activity relationships not apparent from single-compound studies
Mikus et al., 2001
The FAK-Paxillin Connection Beyond VEGF, BPC-157 activates the FAK-paxillin system that drives cell migration to injury sites
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Studies of microbial colonization in sterile mice have shown that increased platelet reactivity and thrombosis potential were sufficient to be delivered via microbial CutC-dependent TMA/TMAO production in a host (Skye et al., 2018) ( Figure 1D )
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