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This activation induces ferroptosis in mouse lung epithelial (MLE-12) cells, contributing to lung congestion, edema, and inflammation ( Figure 5 ) Moreover, H1N1 infection triggers differential expression of ferroptosis-related genes and metabolites in human nasal epithelial progenitor cells (hNEPCs)
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Nuwayhid said
These results establish the HAGSHPVA scaffold as a redox-active, ECM mimetic nanofibrous platform with strong potential for next-generation antioxidant-enriched wound-healing applications
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