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GLP-1RAs thus prevent the occurrence of retinal damage associated with oxidative-stress-induced activation of glial cells and collectively foster a healthy retinal microenvironment by virtue of preventing cellular overexertion [94, 100]

These data collectively suggest that the observed increase in hepatic protein-SSG formation in HFD-fed animals can result from a combination of both increased S -glutathionylation processes and suppressed de-glutathionylation reactions

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