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To confirm that interface IV is essential for c-MET activation, we introduced Y673A mutation into HGF to disrupt this interface, and showed that HGF Y673 mutant has a great deficiency in binding to c-MET as well as promoting c-MET activation (Fig
BPC-157 is conventionally produced as an acetate salt (BPC-157 acetate)
Abdel Moneim AE, et al
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