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One key process is glutamate excitotoxicity, where a buildup of glutamate at the synapse due to reduced function of the transporter EAAT2 (also known as GLT-1) leads to overstimulation of N-methyl-D-aspartate (NMDA) receptors

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Dehydration can exacerbate compensatory heart rate increases and contribute to orthostatic symptoms

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(2023): Semaglutide and Cardiovascular Outcomes in Obesity Without Diabetes