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41 Galectin-3 has been implicated in the pathophysiology of hepatic inflammation and fibrosis through both intracellular effects (such as antiapoptotic activity and macrophage differentiation) and extracellular functions (acting as a chemokinetic/chemotactic factor)
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An epigenomic study of mCRPC patients identified a subset of CpG Methylator Phenotype (CMP), this is, with a high methylation profile, which harbored exclusive mutations in IDH1 among others (TET2 or BRAF) [201]
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predominantly by rebuilding the extracellular matrix and working to offset effects from damaging pro-inflammatory cytokines through the production of counteractive polyamine compounds ( Infecting mice with the pathogen Mycobacterium tuberculosis (Mtb) promotes FAO in macrophages during lung infection ( Functionally targeting macrophage metabolism has also shown particular promise in anti-tumor therapies
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