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In fact, proinflammatory cytokines are often released during the headache phase of a migraine by the major pain-signaling pathway in the brain, known as the trigeminovascular system
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This signaling becomes particularly important when caloric intake drops significantly
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overexpressed miR-340-5p in the myocardium by direct myocardial injection of AVV-9 containing miR-340-5p precursor, which targeted inhibition of MyD88 and thus restored declining GSH levels, alleviated insult of cardiac function and oxidative injury induced by sepsis in the hearts of mice [253]
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