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The collagenase model replicates the matrix disorganization and degenerative pathology of clinical tendinopathy more closely than transection models, strengthening the relevance of the findings to the human chronic tendinopathy condition

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A peptide that survives the stomach intact still faces intestinal brush-border peptidases and first-pass hepatic metabolism before it can reach systemic circulation

Each has a slightly different receptor profile and pharmacokinetic profile, but all share the core GLP-1 mechanism

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