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doi.org/10.2174/1381612824666180712110447 Seiwerth, S., et al

However, the conversion of GPX4 and 4-HNE and cell death were strikingly inhibited in the GPX4-overexpressing mice

VA and QE equally reduced chromosome abnormalities and aberrant cells in bone marrow implying potentialities in lessening cytogenotoxicity influences of cancer chemotherapy (Chen et al., 2011)

SS-31 directly targets the mitochondrial dysfunction that is a primary mechanism of taxane and platinum-based chemotherapy neurotoxicity

Enriched with PHA to gently remove dead skin cells