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Dihexa's synaptogenic effects require activation of the c-Met receptor, as demonstrated by experiments using c-Met-specific short hairpin RNA (shRNA) which completely abolished Dihexa's capacity to induce spine formation

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In addition, clinical evidence suggests the drugs elevate the risk of pancreatitis, pancreatic cancer and gallbladder problems and adversely affect the bile ducts in the liver

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Keshavarz-Bahaghighat, M.R

These results indicate that MTX treatment delays cell cycle progression partly by arresting S phase