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This alteration leads to an increase in primary and conjugated bile acids (e.g., cholic acid, taurocholate, and glycocholic acid, chenodeoxycholic acid) and a decrease in secondary bile acids

(25767408, 21970774) Toxicity from acetylcysteine is therefore clearly possible

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In contrast, in glucose-starved cells, we observed a strong HyPer7 response beginning with the lowest H 2 O 2 concentration tested

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