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The research group posited that GlyNACs effects stemmed from mitigating several hallmarks of aging, including oxidative stress, inflammation, and mitochondria
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doi: 10.3390/biology11020239 214 HansonPMYangRYWuJChenJTLedesmaDTsouSCet al
Mitochondrial-mediated ROS cause oxidative damage to cellular structures, particularly mitochondrial DNA, somatic mutations, genome instability, inactivation of tumor suppressor genes, alterations in metabolic and signaling pathways with simultaneous activation of compensatory antioxidant mechanisms, all of which contribute to cell transformation and thus promote various types of cancer [42]
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