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GSH deficiency has a large negative impact on the mitochondrial respiration machinery, slowing down the TCA cycle at the ETC branch point (i.e

Chait A, den Hartigh LJ

Biological Psychiatry: Cognitive Neuroscience and Neuroimaging, 5, 163-172

However, because it is inactive, the liver must expend cellular energy to strip away the cyanide molecule (decyanation) before the body can even begin to use it

Small numbers of CD4+ T cells can induce development of lymphedema

Thus, liver damage is quite sensitive to polymorphisms in the glucoronosyl transferases and this probably explains some of the variation in patient response