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The levels of soluble transferrin receptor (sTFR), a fragment of TFR1 on the cell membrane secreted into the circulation, were obviously higher in the blood and urine of IgAN patients than in normal individuals [267, 273]

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Nrf2 depletion in macrophages leads to increased foam cell formation, increases the M1 inflammatory phenotype with enhanced expression of pro-inflammatory monocyte chemoattractant protein-1 and interleukin-6, and aggravates atherosclerosis (244, 245)

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Unlike oral supplements, glutathione infusion therapy ensures rapid absorption for visible results