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While current research confirms that macrophage ferroptosis promotes atherosclerosis progression and that hepcidin influences atherosclerotic processes by regulating iron content, the precise relationship between macrophage ferroptosis and hepcidin remains unestablished
Next, we treated HepG2 cells with BCNU, a chemical inhibitor of GR, the enzyme converting GSSG back into GSH
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Energy Environ Sci
doi: 10.1007/BF02030347 35 YazarMSarbanSKocyigitAIsikanUE
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