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The main mechanism of the therapeutic effect of GLP-1R agonists in obesity is considered to be via reduced calorie intake, and this is also considered to be true for their therapeutic effects on MASH 2

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With the assistance of methionine adenosyltransferase(MAT), methionine is transformed into S-adenosylmethionine (SAM), a molecule [67] involved in epigenetic gene expression and cellular differentiation through its involvement in methylation [68, 69]

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