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Here we show that under our experimental conditions in vitro, HGF adopts an enhanced form with dynamically increased receptor-binding affinity and nitration-dysfunction resistance through interaction with lipoic acid trisulfide (LASSS)

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This copper-peptide bond produces the compound's biological effects, triggering gene expression changes that promote anti-aging and tissue repair responses

Furthermore, L-SOD could also reduce OxS to intestinal barrier, thereby ameliorating the vicious circle between hyperglycemia and the oxidative damage 751

PLOS Genet