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This results from GLP-1 receptor activation in the brainstem's chemoreceptor trigger zone, as well as delayed gastric emptying
Experimental data also suggest anti-inflammatory effects relevant to diabetic kidney injury: GLP-1 signaling can suppress AGERAGE pathways, leading to downstream reductions in endothelial vascular cell adhesion molecule 1 (VCAM-1) expression and mesangial monocyte chemoattractant protein 1 (MCP1) induction, alongside shifts toward an anti-inflammatory (M2-like) macrophage phenotype in the renal microenvironment [3]
S., Christensen, S
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