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The weight lost on these medications does not stay off once the medication stops

[1] [2] It works by mimicking the incretin hormone GLP-1, which stimulates insulin secretion in a glucose-dependent manner, suppresses glucagon release, slows gastric emptying, and reduces appetite through central nervous system pathways

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Mice in the GHK-Cu group had an enhanced performance on special memory tests and learning navigation tasks and demonstrated decreased neuroinflammation and axonal damage markers as compared to the placebo group

Mitochondrial turnover is controlled by signaling molecules such as Bcl-2, which inhibit apoptosis and BAD/BAX signaling, which induces apoptosis and mitophagy

But here's something many people don't realize: GLP-1 medications don't replace healthy eating habitsthey make them easier to follow