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Mechanistically, NNMT suppression increased adipose SAM and NAD, upregulated polyamine flux enzymes (ODC, SSAT), and altered histone methylation consistent with a shift toward oxidative metabolism [2]

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Abstract Type 1 diabetes (T1D) is a complex autoimmune disorder that leads to the destruction of insulin-producing pancreatic beta cells

The formation of osteoclasts is regulated by M-CSF and receptor activator of nuclear factor kappa- ligand (RANKL)

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