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It also facilitates DNA synthesis and repair, stabilizes cellular membranes, and regulates appropriate immune responses (5-7)
No major adverse effects reported in preclinical models
281 Importantly, while acute or overwhelming endoplasmic reticulum stress can induce cell death, chronic UPR activation favors drug tolerance and persistence, positioning endoplasmic reticulum stress adaptation as a systems-level resistance mechanism complementary to oncogenic signaling network adaptation
therefore, it does not appear to trigger diabetes development or induce glucose intolerance in test models, though further studies are necessary to explore this aspect
Centrifuge anticoagulant-treated blood at 1,000 g for 10 min at 4C
was treating workers at a battery factory for lead poisoning when he noticed that some of his patients had improved angina pectoris following chelation therapy
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