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doi: 10.1016/j.ijid.2020.03.017 300 YuanS.JiangS.-C.ZhangZ.-W.FuY.-F.HuJ.LiZ.-L

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Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired

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Histologically, epidermal thickening, severe hyperkeratosis, and epidermal acanthosis were seen (de Juanes et al., 2009)