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Metabolic pathways and immunometabolism in rare kidney diseases

37 In our study, we found that Clopidogrel downregulated the expression of phosphorylated eNOS and induced ER stress in the rat gastric mucosa cells and the BPC 157 protected against Clopidogrel-induced gastric mucosa dysfunction by alleviating ER stress and promoting eNOS -mediated angiogenesis whereas the effect of L-NAME is completely the opposite

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doi: 10.1186/s13148-020-00837-1 296 GilbertWVBellTASchaeningC