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Genomics 7, 291301
(2013) 8:e69827
In the present study, liraglutide effectively inhibited PFKFB3-driven glycolysis in TGF-1 + IL-1-stimulated fibroblasts and silica-induced fibrotic lungs, as well as the enhancement of mitochondrial oxidative phosphorylation in fibroblasts, indicating that activating GLP-1R was sufficient to maintain metabolic homeostasis during pulmonary fibrosis
Mitochondria play an important role in the generation of metabolic energy in form of ATP necessary for the cell survival and proper functioning, disruption of mitochondrial function in the brain result in the reduction of ATP levels, impairment of all energy-dependent cellular functions, disturbance of the generation of biosynthetic precursors leading to neurological and neurodevelopmental disorders [29]
Imaging 42, 14311440
Semaglutide onset of action for metabolic effects is well documented, though anti-inflammatory timelines may differ
