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Similarly, Jia et al (91) reported that elevated neuronal lactate uptake augments ROS production and mitochondrial oxidative metabolism, thereby disrupting the balance between ROS generation and detoxification, impairing ATP synthesis and ultimately leading to peripheral axonal degeneration
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We conclude that our LCMS/MS-based quantitative analyses for acyl group- and site-selectivity of HDAC isoforms on acylated nucleosomes are powerful tools for understanding how histone acylations are regulated, which enables to gain new insights into the fundamental epigenetic mechanisms
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